Stroke, Vol 15, 359-362, Copyright © 1984 by American Heart Association
R Kurlan, RL Krall and JA Deweese
Patients who have undergone a Blalock-Taussig anastomosis for treatment of
congenital heart disease may have the vascular anatomy of the subclavian
steal syndrome. Cerebral ischemia has been reported in such patients, but
not when total surgical correction has eliminated other predisposing
factors. We report a patient who developed vertebrobasilar insufficiency 31
years after Blalock-Taussig anastomosis and 4 years after total
intracardiac repair of tetralogy of Fallot. He had angiographically proven
subclavian steal and no other known predisposing factor for cerebral
ischemia. This case suggests that symptomatic subclavian steal may be a
late risk of surgical treatment of congenital heart disease that leaves the
vascular anatomy of subclavian steal intact. Vascular reconstructive
surgery can be effective treatment for these patients and may be indicated
prophylactically at the time of intracardiac repair if subclavian steal
syndrome becomes a more frequently recognized sequela of prior Blalock-
Taussig anastomosis.
ARTICLES
Vertebrobasilar ischemia after total repair of tetralogy of Fallot: significance of subclavian steal created by Blalock-Taussig anastomosis. Vertebrobasilar ischemia after correction of tetralogy of Fallot
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