Donate Help Contact The AHA Sign In Home
American Heart Association
Stroke
Search: search_blue_button Advanced Search
This Article
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow Request Permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Alarcon, F.
Right arrow Articles by Duenas, G.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Alarcon, F.
Right arrow Articles by Duenas, G.

Stroke, Vol 23, 224-228, Copyright © 1992 by American Heart Association


ARTICLES

Cerebral cysticercosis and stroke

F Alarcon, F Hidalgo, J Moncayo, I Vinan and G Duenas
Department of Neurology, Eugenio Espejo Hospital, Quito, Ecuador.

BACKGROUND AND PURPOSE: In 1985 we initiated a protocol for examining the relationship between cerebral cysticercosis and stroke. METHODS: In 420 stroke patients admitted to our department, our standard protocol of tests included blood tests, cardiac investigations, angiography, and immunologic cerebrospinal fluid measures. We assessed the following possible risk factors: arterial hypertension, diabetes, cardiopathy, high levels of cholesterol and triglycerides, smoking, alcohol abuse, and cerebral cysticercosis. RESULTS: Of the 420 patients with stroke, we found cerebral cysticercosis in 31, five of whom were greater than 65 years of age and 26 of whom were less than or equal to 65 years. We determined that cerebral cysticercosis was the only possible risk factor for stroke in one of the five older patients and 15 of the 26 younger and middle-aged patients. Cortical infarctions were found in five of the 31 patients, with cerebral cysticercosis and lacunar infarctions in nine of these patients. One patient had intracystic hemorrhage. In 16 cases, neurological deficit was related to single or multiple cysts, colloids, granulomas, diffuse lesions, or pericystic edema. All patients with cerebral cysticercosis quickly recovered from their neurological deficit, except one who had a hemorrhagic cyst and died and another who remained disabled. CONCLUSIONS: We established that, in patients with neurocysticercosis, occlusion of the small cortical or penetrating vessels at the base of the brain caused by arteriopathy was the most common mechanism of the stroke. Moreover, there is a probable association between cerebral cysticercosis and the susceptibility to stroke, particularly among young and middle-aged patients.


This article has been cited by other articles:


Home page
J Child NeurolHome page
A. Bouldin and J. D. Pinter
Resolution of Arterial Stenosis in a Patient With Periarterial Neurocysticercosis Treated With Oral Prednisone
J Child Neurol, December 1, 2006; 21(12): 1064 - 1067.
[Abstract] [PDF]


Home page
J. Neurol. Neurosurg. PsychiatryHome page
F Alarcon, J C M Zijlmans, G Duenas, and N Cevallos
Post-stroke movement disorders: report of 56 patients
J. Neurol. Neurosurg. Psychiatry, November 1, 2004; 75(11): 1568 - 1574.
[Abstract] [Full Text] [PDF]


Home page
NEJMHome page
P. F. Finelli, F. S. Buonanno, and M. P. Frosch
Case 16-2002: Neurocysticercosis
N. Engl. J. Med., October 3, 2002; 347(14): 1117 - 1117.
[Full Text] [PDF]


Home page
StrokeHome page
O. Y. Bang, J. H. Heo, S. A. Choi, and D. I. Kim
Large Cerebral Infarction During Praziquantel Therapy in Neurocysticercosis
Stroke, January 1, 1997; 28(1): 211 - 213.
[Abstract] [Full Text]