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(Stroke. 2005;36:e45.)
© 2005 American Heart Association, Inc.
Short Communications |
From the Departments of Neurosurgery (J.E.C., F.U.), Division of Neuroendovascular Surgery and Interventional Neuroradiology (J.E.C., M.G.), and Neurology (T.B.H.), Hadassah-Hebrew University Medical Center, Jerusalem, Israel.
Correspondence to José E. Cohen, MD, Department of Neurosurgery, Hadassah University Medical Center, Ein-Kerem, P.O. Box 12,000, Jerusalem 91120, Israel. E-mail jcohenns{at}yahoo.com
Background and Purpose The prognosis of traumatic dissection of the internal carotid artery is worse than for spontaneous dissections. Rapid stenting followed by antiplatelet therapy may prevent complications when anticoagulation therapy is not applicable.
Methods Patients with angiographically proven traumatic carotid artery dissection and hemodynamic significant hemispheric hypoperfusion, or in whom anticoagulant therapy was either contraindicated or failed clinically, were regarded as being at high risk for stroke and were selected for stenting.
Results Ten patients with traumatic dissection underwent stenting. Endovascular treatment reduced mean dissection stenosis from 69% to 8%. During a mean clinical follow-up time of 16 months, none had additional transient ischemic attacks or stroke. Doppler ultrasound studies did not detect any signs of de novo in-stent stenosis.
Conclusion In selected cases of traumatic carotid artery dissections, endovascular stent-assisted angioplasty immediately restored the integrity of the vessel lumen and prevented efficiently the occurrence of new ischemic events, without additional anticoagulation.
Key Words: carotid arteries stents stroke, ischemic trauma
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