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(Stroke. 2007;38:407.)
© 2007 American Heart Association, Inc.

Research Reports

Insular Cortex Hypoperfusion and Acute Phase Blood Glucose After Stroke

A CT Perfusion Study

From Division of Clinical Neurosciences, University of Glasgow, Institute of Neurological Sciences, Southern General Hospital, Glasgow, Scotland, UK.

Correspondence to Dr Keith Muir, Division of Clinical Neurosciences, University of Glasgow, Institute of Neurological Sciences, Southern General Hospital, Glasgow G51 4TF, Scotland, UK. E-mail k.muir{at}clinmed.gla.ac.uk

Background and Purpose— Insular cortex ischemia is proposed to mediate a sympathetic stimulus that leads to acute hyperglycemia after stroke.

Methods— We retrospectively analyzed insular perfusion on perfusion CT (median 180 minutes after onset) in 35 patients.

Results— We found no association of hypoperfusion (relative cerebral blood flow <0.51) with early (<6 hours) or delayed (<72 hours) hyperglycemia, or hemispheric lateralization.

Conclusions— Insular cortex hypoperfusion <6 hours after stroke onset was not associated with hyperglycemia.

Key Words: blood glucose • cerebral blood flow • hyperglycemia • insula • perfusion CT

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