Stroke, Vol 7, 626-631, Copyright © 1976 by American Heart Association
PC Burger, JG Burch and U Kunze
A 51-yearold man with moderate intermittent hypertension had a rapidly
progressive, profound dementia in the absence of significant localizing
neurological signs. Postmortem examination disclosed the vascular
alterations and diffuse white matter degeneration which characterize
subcortical arteriosclerotic encephalopathy (SAE) or Binswanger's disease.
The case underscores the need to consider vascular disease as an etiology
of dementia -- even in the absence of focal neurological deficit.
ARTICLES
Subcortical arteriosclerotic encephalopathy (Binswanger's disease). A vascular etiology of dementia
This article has been cited by other articles:
![]() |
J.-X. Lin, H. Tomimoto, I. Akiguchi, A. Matsuo, H. Wakita, H. Shibasaki, and H. Budka Vascular Cell Components of the Medullary Arteries in Binswanger's Disease Brains : A Morphometric and Immunoelectron Microscopic Study Stroke, August 1, 2000; 31(8): 1838 - 1842. [Abstract] [Full Text] [PDF] |
||||
![]() |
D. J. Libon, M. Scanlon, R. Swenson, and H. Branch Coslet Binswanger's Disease: Some Neuropsychological Considerations J Geriatr Psychiatry Neurol, January 1, 1990; 3(1): 31 - 40. [Abstract] [PDF] |
||||
![]() |
W. A. Marks, R. M. Shuman, R. W. Leech, and R. A. Brumback Cerebral Degenerations Producing Dementia: Importance of Neuropathologic Confirmation of Clinical Diagnoses J Geriatr Psychiatry Neurol, October 1, 1988; 1(4): 187 - 198. [Abstract] [PDF] |
||||
|
Stroke Home | Subscriptions | Archives | Feedback | Authors | Help | AHA Journals Home | Search Copyright © 1976 American Heart Association, Inc. All rights reserved. Unauthorized use prohibited. |