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Stroke, Vol 15, 157-159, Copyright © 1984 by American Heart Association
EW Massey, A Heyman, C Utley, C Haynes and J Fuchs
During the past seven years 347 patients have been entered into a data bank
at the Duke University Medical Center for evaluation of transient
neurologic ischemia. One hundred fifty eight of these patients had carotid
endarterectomies of whom 24 (15.1%) developed 26 (16.4%) peripheral cranial
nerve palsies. Injury to the peripheral portion of the hypoglossal nerve
was noted in 13 patients, to the cervical branch of the facial nerve in
five and to the recurrent laryngeal nerve branch of the vagus in eight.
Complete recovery of nerve function usually occurred within four months but
residual deficit was present at one year in two patients with facial nerve
and four with hypoglossal nerve involvement. Even though these
complications of carotid endarterectomy are generally benign and transient,
the frequency of occurrence can be reduced if careful attention is given to
anatomic localization of the cranial nerves during surgery.
ARTICLES
Cranial nerve paralysis following carotid endarterectomy
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